This discussion is about what scientific research is needed to further develop theories of collapse and correction
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Correlation between skull/nasomaxillary bone volume and malocclusion/dystonia/TMJ/Apnea etc symptoms
In the Whole Body Breathing theory, the skull is undergrown / growth is incomplete. The prevailing mainstream theory is that growth is complete and the shape is determined by genetics, with some functional component. The orthotropics/functional viewpoint is that growth is complete but the direction of growth/shape of growth is downwards rather than up and forward. One of these viewpoints must be validated to move forward. A study / data review which establishes whether those with crowded teeth, OSA etc issues have less bone volume ie less bone growth compared to those without such issues could determine if the issue is really lack of growth or growth in the wrong direction.
In the Whole Body Breathing theory, the skull is undergrown / growth is incomplete. The prevailing mainstream theory is that growth is complete and the shape is determined by genetics, with some functional component. The orthotropics/functional viewpoint is that growth is complete but the direction of growth/shape of growth is downwards rather than up and forward. One of these viewpoints must be validated to move forward. A study / data review which establishes whether those with crowded teeth, OSA etc issues have less bone volume ie less bone growth compared to those without such issues could determine if the issue is really lack of growth or growth in the wrong direction.
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Skull growth timeline and suture fusion timeline
There are conflicting theories regarding when skull growth is complete (ranging from 18 to 25, and then into the idea that the growth never fully stops), as well as conflicting theories about whether skull suture closure is truly a natural part of growth or if it is a pathology/structural issue. The age at which sutures typically display closing is also not studied heavily enough, with many outlier cases indicating that sutures may remain patent decades past when it is assumed they close (early adulthood). The extent to which sutures can re-open, and what growth potential even a partially closed suture or junction can have/re-activate is crucial to determining how much correction is possible once someone has deviated from proper growth.
There are conflicting theories regarding when skull growth is complete (ranging from 18 to 25, and then into the idea that the growth never fully stops), as well as conflicting theories about whether skull suture closure is truly a natural part of growth or if it is a pathology/structural issue. The age at which sutures typically display closing is also not studied heavily enough, with many outlier cases indicating that sutures may remain patent decades past when it is assumed they close (early adulthood). The extent to which sutures can re-open, and what growth potential even a partially closed suture or junction can have/re-activate is crucial to determining how much correction is possible once someone has deviated from proper growth.
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Role of genetics in improper growth
The genetic component of improper growth/malocclusion has long been the hand-waved answer regarding why modern humans are in a state of craniofacial collapse. However no solid study about specific genes, markers, gene combinations etc has been done to prove this theory. This is not how evidence based medicine should work. Stuctural collapse to life-threatening levels occurs within 1-2 generations even when parents/grandparents are well developed and within the same genetic pool: This makes the genetic theory less plausible. If the genetic component can be determined, then it can be worked on and researched.
The genetic component of improper growth/malocclusion has long been the hand-waved answer regarding why modern humans are in a state of craniofacial collapse. However no solid study about specific genes, markers, gene combinations etc has been done to prove this theory. This is not how evidence based medicine should work. Stuctural collapse to life-threatening levels occurs within 1-2 generations even when parents/grandparents are well developed and within the same genetic pool: This makes the genetic theory less plausible. If the genetic component can be determined, then it can be worked on and researched.
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Dietary Componants
A dietary component (or multiple dietary components) being missing during development has long been theorized as the cause for poor development. Although much advancement has occurred in nutritional science and further developments are being made, theories about certain diets leading to better/worse outcomes remain unproven, ancedotal, and speculatory, with many counter examples for any given idea.
A dietary component (or multiple dietary components) being missing during development has long been theorized as the cause for poor development. Although much advancement has occurred in nutritional science and further developments are being made, theories about certain diets leading to better/worse outcomes remain unproven, ancedotal, and speculatory, with many counter examples for any given idea.
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Lowered infant/childhood mortality and selection bias
The structural / facial collapse makes a person less resiliant to all causes of mortality. Disease, pathogens, physical threats may all be more harmful to a child who is already in a state of structural collapse and compensation. The mental state is also affected. As antibiotics and other modern treatments are bringing large portions of the population to adulthood who otherwise may have died in childhood, it may be plausable that in previous generations that undergrown/improperly grown children did not make it to adulthood and we are seeing only the well developed examples when looking at adults of recent history. In more wealthy stratas of society the collapse of facial form seems to be even more evident at an earlier time than in the general population; The collapse of human jaws around the time when settled agriculture became the norm etc may all indicate that as it became easier for children with improper growth to survive into adulthood due to societal support, improper growth became prevalant (and especially prevalant once childhood mortality dropped in the past 1-200 years)
The structural / facial collapse makes a person less resiliant to all causes of mortality. Disease, pathogens, physical threats may all be more harmful to a child who is already in a state of structural collapse and compensation. The mental state is also affected. As antibiotics and other modern treatments are bringing large portions of the population to adulthood who otherwise may have died in childhood, it may be plausable that in previous generations that undergrown/improperly grown children did not make it to adulthood and we are seeing only the well developed examples when looking at adults of recent history. In more wealthy stratas of society the collapse of facial form seems to be even more evident at an earlier time than in the general population; The collapse of human jaws around the time when settled agriculture became the norm etc may all indicate that as it became easier for children with improper growth to survive into adulthood due to societal support, improper growth became prevalant (and especially prevalant once childhood mortality dropped in the past 1-200 years)